₁Keiko Kagami

Summary for 2003 [at-glance overview of Keiko Kagami work for the selected year as suggested by data mining algorithms]

This is an Authoratory overview of author Keiko Kagami. According to available data, in 2003 Keiko Kagami published at least 1 articles. This work was completed in collaboration with several authors including Yagita, Hideo and Okumura, Ko. Many other authors have collaborated with Keiko Kagami as well. There is no information about the funding or the grants awarded to support this work.

Analysis of the article abstracts and the titles suggests that Keiko Kagami professional interests are focused around "apoptosis-inducing ligand trail", "leukemia cell lines" and "ligand trail frequently". These might also be referred to as "cell death", "cell lines" or "leukemia cells". Statistical analysis shows that Keiko Kagami's writing is likely to contains terms "trail", "cell", "leukemia", "death", "ph1-positive" and "fasl".

The majority of Keiko Kagami articles are affiliated with Department of Pediatrics, School of Medicine, University of Yamanashi, Nakakoma, Japan. Sadly, no email addresses were found for any of the affiliations.

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Alternative names (1) [other authors with names similar to Keiko Kagami]

₁₀Kagami, K

Year 2003 Charts [Keiko Kagami frequent coauthors and the most common unique vocabulary terms for the selected year]

Historical Performance Charts [graphical overview of Keiko Kagami publication and funding history for the range of years]

Keiko Kagami Coauthors (13) [all coauthors with the number of joint publications]

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Affiliations (1) [condensed summary of the Keiko Kagami affiliations, the leading number and the color conveys importance]

₁Department of Pediatrics, School of Medicine, University of Yamanashi, Nakakoma, Japan.

Key Opinion Leader In [Keiko Kagami is considered a key opinion leader in any keyword where the rank of 5 is achieved, the leading number before the keyword shows the author's rank as compared to all other authors]

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Keywords (32) [keywords assigned to Keiko Kagami articles by PubMed, the leading number and the color conveys importance]

₁Apoptosis | ₁Receptors, Tumor Necrosis Factor | ₁TNF-Related Apoptosis-Inducing Ligand | ₁Receptors, TNF-Related Apoptosis-Inducing Ligand | ₁Arabidopsis Proteins | ₁NF-kappa B | ₁Fas Ligand Protein | ₁Fatty Acid Desaturases | ₁Death Domain Receptor Signaling Adaptor Proteins | ₁Tumor Necrosis Factor-alpha | ₁Tumor Cells, Cultured | ₁Pyrimidines | ₁Leukemia, Lymphocytic, Acute, L2 | ₁Apoptosis Regulatory Proteins | ₁Humans | ₁Recombinant Proteins | ₁Membrane Glycoproteins | ₁Caspase 1 | ₁Carrier Proteins | ₁Leupeptins | ₁Amino Acid Chloromethyl Ketones | ₁Drug Screening Assays, Antitumor | ₁Peptides | ₁Neoplasm Proteins | ₁Enzyme Inhibitors | ₁Leukemia, Myeloid, Philadelphia-Positive | ₁Piperazines | ₁CASP8 and FADD-Like Apoptosis Regulating Protein | ₁Intracellular Signaling Peptides and Proteins | ₁Neoplastic Stem Cells | ₁Drug Resistance, Neoplasm | ₁Fusion Proteins, bcr-abl

Keiko Kagami Unique Vocabulary (96) [single words, word pairs and phrases obtained by analysis of abstracts and titles, the leading number and the color conveys importance; Keiko Kagami might be a good expert witness on these terms]

₁₁trail | ₈cell | ₇leukemia | ₆death | ₆ph1-positive | ₄fasl | ₄lines | ₄ligand | ₃cells | ₃apoptosis-inducing | ₃philadelphia | ₃caspase-8 | ₃protein | ₂possible | ₂acute | ₂induces | ₂apoptosis | ₂immunotherapy | ₂sensitivity | ₂chromosome-positive | ₂factor | ₂frequently | ₂leukemias | ₂tnf-related | ₂contribution | ₂death-inducing | ₂leukemia-derived | ₂expressed | ₂expression | ₂dr4 | ₂dr5 | ₁negative
₄cell death | ₄cell lines | ₃leukemia cells | ₃trail fasl | ₃leukemia cell | ₃apoptosis-inducing ligand | ₃ligand trail | ₂frequently induces | ₂trail frequently | ₂chromosome-positive leukemia | ₂immunotherapy ph1-positive | ₂philadelphia chromosome-positive | ₂ph1-positive leukemias | ₂induces apoptosis | ₂possible contribution | ₂apoptosis philadelphia | ₂tnf-related apoptosis-inducing | ₂dr4 dr5 | ₂ph1-positive leukemia | ₁cells tumor | ₁expressed ubiquitously | ₁chronic myelogenous | ₁fadd caspase-8 | ₁flip flice | ₁imatinib mesylate | ₁enzyme caspase-8 | ₁moreover | ₁ubiquitously ph1-positive | ₁primary leukemia | ₁leukemias tnf-related | ₁sensitivity philadelphia | ₁trail fas
₃apoptosis-inducing ligand trail | ₃leukemia cell lines | ₂ligand trail frequently | ₂philadelphia chromosome-positive leukemia | ₂tnf-related apoptosis-inducing ligand | ₂chromosome-positive leukemia cells | ₂immunotherapy ph1-positive leukemias | ₂apoptosis philadelphia chromosome-positive | ₂frequently induces apoptosis | ₂induces apoptosis philadelphia | ₂trail frequently induces | ₂ph1-positive leukemia cell | ₁death caspase-dependent enhanced | ₁cell lines refractory | ₁cells notably trail | ₁dr4 dr5 trail-induced | ₁trail fas ligand | ₁apoptotic cell death | ₁possible contribution endogenously | ₁moreover | ₁lines tnf-related apoptosis-inducing | ₁fasl depending dr4 | ₁cell lines trail | ₁contribution endogenously expressed | ₁ph1-positive acute lymphoblastic | ₁ph1 positive leukemia | ₁depending dr4 dr5 | ₁contribution molecules immunotherapy | ₁interleukin-1-converting enzyme caspase-8 | ₁cell-surface expression death-inducing | ₁dr5 trail-induced cell | ₁related apoptosis-inducing ligand

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