₄Robert W Schnepp

Summary for 2004 [at-glance overview of Robert W Schnepp work for the selected year as suggested by data mining algorithms]

This is an Authoratory overview of author Robert W Schnepp. According to available data, in 2004 Robert W Schnepp published at least 4 articles. This work was completed in collaboration with several authors including Hua, Xianxin and Hou, Zhaoyuan. Several other authors have collaborated with Robert W Schnepp as well. There is no information about the funding or the grants awarded to support this work.

Robert W Schnepp has rank 160 when compared to all other authors using the keyword Proto-Oncogene Proteins. Analysis of the article abstracts and the titles suggests that Robert W Schnepp professional interests are focused around "tumor suppressor menin", "complementation menin-null cells" and "neoplasia type men1". These might also be referred to as "cell proliferation", "tumor suppressor" or "suppressor menin". Statistical analysis shows that Robert W Schnepp's writing is likely to contains terms "menin", "men1", "tumor", "cell", "proliferation" and "suppressor".

The majority of Robert W Schnepp articles are affiliated with Abramson Family Cancer Research Institute, Department of Cancer Biology and Signal Transduction Program, University of Pennsylvania, Philadelphia, Pennsylvania 19104-6160, USA. Sadly, no email addresses were found for any of the affiliations.

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Alternative names (0) [other authors with names similar to Robert W Schnepp]

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Year 2004 Charts [Robert W Schnepp frequent coauthors and the most common unique vocabulary terms for the selected year]

Historical Performance Charts [graphical overview of Robert W Schnepp publication and funding history for the range of years]

Robert W Schnepp Coauthors (8) [all coauthors with the number of joint publications]

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Affiliations (1) [condensed summary of the Robert W Schnepp affiliations, the leading number and the color conveys importance]

₄Abramson Family Cancer Research Institute, Department of Cancer Biology and Signal Transduction Program, University of Pennsylvania, Philadelphia, Pennsylvania 19104-6160, USA.

Key Opinion Leader In [Robert W Schnepp is considered a key opinion leader in any keyword where the rank of 5 is achieved, the leading number before the keyword shows the author's rank as compared to all other authors]

₁₆₀Proto-Oncogene Proteins | ₆₈₅Cell Division

Keywords (32) [keywords assigned to Robert W Schnepp articles by PubMed, the leading number and the color conveys importance]

₄Proto-Oncogene Proteins | ₃Cell Division | ₃Mice | ₃Animals | ₃Humans | ₃Cells, Cultured | ₃Cell Line | ₂Annexin A5 | ₂Blotting, Western | ₂Plasmids | ₂Apoptosis | ₂Genetic Complementation Test | ₂Up-Regulation | ₂DNA | ₂Flow Cytometry | ₂Mutation | ₂Enzyme Inhibitors | ₂Fibroblasts | ₂Multiple Endocrine Neoplasia Type 1 | ₂Cell Separation | ₁Mutagenesis, Site-Directed | ₁Mutagenesis | ₁Retroviridae | ₁Gene Expression Regulation, Neoplastic | ₁bcl-2 Homologous Antagonist-Killer Protein | ₁Adenoviridae | ₁Cell Proliferation | ₁Tumor Necrosis Factor-alpha | ₁Cell Cycle | ₁DNA-Binding Proteins | ₁RNA, Messenger | ₁Nucleosomes

Robert W Schnepp Unique Vocabulary (96) [single words, word pairs and phrases obtained by analysis of abstracts and titles, the leading number and the color conveys importance; Robert W Schnepp might be a good expert witness on these terms]

₄₅menin | ₁₅men1 | ₁₄tumor | ₁₄cell | ₁₂proliferation | ₁₀suppressor | ₁₀dna | ₉apoptosis | ₈cells | ₈binding | ₈expression | ₇gene | ₇igfbp-2 | ₇endocrine | ₇multiple | ₆menin-null | ₆kinase | ₅complementation | ₅nuclear | ₅ask | ₄mutated | ₄patients | ₄repression | ₄type | ₄show | ₄neoplasia | ₃encodes | ₃activator | ₃s-phase | ₃development | ₃interaction | ₃disease-related
₁₂cell proliferation | ₁₀tumor suppressor | ₇suppressor menin | ₇multiple endocrine | ₆menin-null cells | ₅complementation menin-null | ₄binding dna | ₄endocrine neoplasia | ₄neoplasia type | ₄type men1 | ₄igfbp-2 gene | ₃s-phase kinase | ₃men1 patients | ₃mutated men1 | ₃activator s-phase | ₃syndrome characterized | ₃promoter igfbp-2 | ₃essential repression | ₃menin essential | ₃regulates expression | ₃unclear menin | ₃induces apoptosis | ₃tumor syndrome | ₃embryonic fibroblasts | ₃whereas complementation | ₃cells menin | ₃menin interacts | ₃murine embryonic | ₃wild-type menin | ₃menin regulates | ₃apoptosis murine | ₂localization signals
₇tumor suppressor menin | ₅complementation menin-null cells | ₄neoplasia type men1 | ₄endocrine neoplasia type | ₄multiple endocrine neoplasia | ₃activator s-phase kinase | ₃mutated men1 patients | ₃apoptosis murine embryonic | ₃menin essential repression | ₃tumor syndrome characterized | ₃menin-null cells menin | ₃murine embryonic fibroblasts | ₃whereas complementation menin-null | ₃induces apoptosis murine | ₃menin regulates expression | ₂nuclear localization signals | ₂characterized development tumors | ₂suppressor menin activator | ₂direct binding dna | ₂expression insulin-like growth | ₂multiple endocrine organs | ₂functional interaction tumor | ₂gene mutated men1 | ₂insulin-like growth factor | ₂factor binding protein | ₂menin activator s-phase | ₂encodes nuclear protein | ₂patients men1 encodes | ₂men1 patients men1 | ₂binding dna tumor | ₂syndrome characterized development | ₂development tumors multiple

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