₇Eduard F Stange

Summary for 2004 [at-glance overview of Eduard F Stange work for the selected year as suggested by data mining algorithms]

This is an Authoratory overview of author Eduard F Stange. According to available data, in 2004 Eduard F Stange published at least 7 articles. This work was completed in collaboration with several authors including Fellermann, Klaus and Herrlinger, Klaus R. Many other authors have collaborated with Eduard F Stange as well. There is no information about the funding or the grants awarded to support this work.

Eduard F Stange has rank 42 when compared to all other authors using the keyword Crohn Disease. Analysis of the article abstracts and the titles suggests that Eduard F Stange professional interests are focused around "inflammatory bowel disease", "bactericidal permeability-increasing protein" and "escherichia coli nissle". These might also be referred to as "coli nissle", "inflammatory bowel" or "crohn disease". Statistical analysis shows that Eduard F Stange's writing is likely to contains terms "disease", "coli", "hbd-2", "protein", "patients" and "nissle".

Unfortunately, there is not enough information to establish Eduard F Stange affiliation. Luckily, several email addresses are on file.

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Alternative names (1) [other authors with names similar to Eduard F Stange]

₆₆Stange, E F

Year 2004 Charts [Eduard F Stange frequent coauthors and the most common unique vocabulary terms for the selected year]

Historical Performance Charts [graphical overview of Eduard F Stange publication and funding history for the range of years]

Emails (2) [email addresses extracted from Eduard F Stange affiliations]

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Eduard F Stange Coauthors (17) [all coauthors with the number of joint publications]

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Affiliations (5) [condensed summary of the Eduard F Stange affiliations, the leading number and the color conveys importance]

₂Department of Internal Medicine I, Robert Bosch Hospital, Stuttgart, Germany.
₁Department of Rheumatology, University of Luebeck, Bad Bramstedt, Germany.
₁Department of Internal Medicine, Robert-Bosch-Hospital, Stuttgart, Germany.
₁Robert-Bosch-Krankenhaus, Zentrum Innere Medizin, Gastroenterologie, Hepatologie und Endokrinologie, Stuttgart.
₁Division of Gastroenterology, Medical University of Schleswig-Holstein Campus Lübeck, D-23538 Lübeck, Germany.

Key Opinion Leader In [Eduard F Stange is considered a key opinion leader in any keyword where the rank of 5 is achieved, the leading number before the keyword shows the author's rank as compared to all other authors]

₄₂Crohn Disease | ₅₉Colitis, Ulcerative

Keywords (32) [keywords assigned to Eduard F Stange articles by PubMed, the leading number and the color conveys importance]

₆Humans | ₄Crohn Disease | ₃Colitis, Ulcerative | ₃Female | ₃Male | ₂Adolescent | ₂Escherichia coli | ₂Inflammatory Bowel Diseases | ₂Gene Expression Regulation | ₂Liver | ₁Base Sequence | ₁C-Reactive Protein | ₁Intestines | ₁Sensitivity and Specificity | ₁beta-Defensins | ₁Defensins | ₁p38 Mitogen-Activated Protein Kinases | ₁CCAAT-Enhancer-Binding Proteins | ₁Hygiene | ₁Tumor Necrosis Factor-alpha | ₁DNA-Binding Proteins | ₁Triglycerides | ₁RNA, Messenger | ₁Cholesterol | ₁Epithelial Cells | ₁Mice, Inbred AKR | ₁Pneumonia, Bacterial | ₁Gastrointestinal Agents | ₁Leucine Zippers | ₁Stress, Psychological | ₁Respiratory Distress Syndrome, Adult | ₁Child

Eduard F Stange Unique Vocabulary (96) [single words, word pairs and phrases obtained by analysis of abstracts and titles, the leading number and the color conveys importance; Eduard F Stange might be a good expert witness on these terms]

₁₃disease | ₉coli | ₉hbd-2 | ₉protein | ₈patients | ₈nissle | ₈cholesterol | ₇bpi-autoantibodies | ₇mice | ₇srebp2 | ₆inflammatory | ₆bowel | ₆crohn | ₆cells | ₆bpi | ₆probiotic | ₆intestinal | ₆induction | ₅bacteria | ₅may | ₅permeability-increasing | ₅infliximab | ₅antibiotic | ₅bactericidal | ₄autoantibodies | ₄nf-kappab | ₄ibd | ₃n-terminal | ₃mucosal | ₃escherichia | ₃effect | ₃activity
₇coli nissle | ₆inflammatory bowel | ₆crohn disease | ₅bowel disease | ₅bactericidal permeability-increasing | ₅permeability-increasing protein | ₄epithelial cells | ₃disease patients | ₃escherichia coli | ₃may contribute | ₃cholesterol synthesis | ₃probiotic bacteria | ₃ulcerative colitis | ₃intestinal epithelial | ₃srebp1 srebp2 | ₃induction hbd-2 | ₂human beta | ₂defensin-2 intestinal | ₂target antigen | ₂ap-1-mediated induction | ₂probiotic bacterium | ₂novel effect | ₂epitope mapping | ₂patients can | ₂hbd-2 promoter | ₂staphylococcal sepsis | ₂antibiotic activity | ₂fatal staphylococcal | ₂igg preparations | ₂innate defense | ₂induction human | ₂autoantibodies bactericidal
₅inflammatory bowel disease | ₅bactericidal permeability-increasing protein | ₃escherichia coli nissle | ₃intestinal epithelial cells | ₃regulatory element binding | ₃cholesterol gallstone-susceptible mice | ₂can impair antibiotic | ₂fatal staphylococcal sepsis | ₂permeability-increasing protein inflammatory | ₂novel effect probiotic | ₂human beta defensin-2 | ₂antibiotic activity bactericidal | ₂sepsis crohn disease | ₂disease patients can | ₂beta defensin-2 intestinal | ₂nissle novel effect | ₂crohn disease infliximab | ₂defensin-2 intestinal epithelial | ₂impair antibiotic activity | ₂protein inflammatory bowel | ₂coli nissle novel | ₂epithelial cells escherichia | ₂ap-1-mediated induction human | ₂autoantibodies bactericidal permeability-increasing | ₂nf-kappab ap-1-mediated induction | ₂effect probiotic bacterium | ₂activity bactericidal permeability-increasing | ₂induction human beta | ₂bowel disease patients | ₂patients can impair | ₂staphylococcal sepsis crohn | ₂cells escherichia coli

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