₄Xianxin Hua

Summary for 2004 [at-glance overview of Xianxin Hua work for the selected year as suggested by data mining algorithms]

This is an Authoratory overview of author Xianxin Hua. According to available data, in 2004 Xianxin Hua published at least 4 articles. This work was completed in collaboration with several authors including Schnepp, Robert W and Hou, Zhaoyuan. Several other authors have collaborated with Xianxin Hua as well. At least 3 different grants are awarded to Xianxin Hua to support this work. All time total of the grant awards on file exceeds $3,127,798.

Xianxin Hua has rank 159 when compared to all other authors using the keyword Proto-Oncogene Proteins. Analysis of the article abstracts and the titles suggests that Xianxin Hua professional interests are focused around "tumor suppressor menin", "complementation menin-null cells" and "neoplasia type men1". These might also be referred to as "cell proliferation", "tumor suppressor" or "suppressor menin". Statistical analysis shows that Xianxin Hua's writing is likely to contains terms "menin", "men1", "tumor", "cell", "proliferation" and "suppressor".

The majority of Xianxin Hua articles are affiliated with Abramson Family Cancer Research Institute, Department of Cancer Biology and Signal Transduction Program, University of Pennsylvania, Philadelphia, Pennsylvania 19104-6160, USA. Sadly, no email addresses were found for any of the affiliations.

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Alternative names (1) [other authors with names similar to Xianxin Hua]

₄₀Hua, X

Year 2004 Charts [Xianxin Hua frequent coauthors and the most common unique vocabulary terms for the selected year]

Historical Performance Charts [graphical overview of Xianxin Hua publication and funding history for the range of years]

Xianxin Hua Coauthors (8) [all coauthors with the number of joint publications]

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Affiliations (1) [condensed summary of the Xianxin Hua affiliations, the leading number and the color conveys importance]

₄Abramson Family Cancer Research Institute, Department of Cancer Biology and Signal Transduction Program, University of Pennsylvania, Philadelphia, Pennsylvania 19104-6160, USA.

Key Opinion Leader In [Xianxin Hua is considered a key opinion leader in any keyword where the rank of 5 is achieved, the leading number before the keyword shows the author's rank as compared to all other authors]

₁₅₉Proto-Oncogene Proteins | ₆₇₉Cell Division

Keywords (32) [keywords assigned to Xianxin Hua articles by PubMed, the leading number and the color conveys importance]

₄Proto-Oncogene Proteins | ₃Cell Division | ₃Mice | ₃Animals | ₃Humans | ₃Cells, Cultured | ₃Cell Line | ₂Annexin A5 | ₂Blotting, Western | ₂Plasmids | ₂Apoptosis | ₂Genetic Complementation Test | ₂Up-Regulation | ₂DNA | ₂Flow Cytometry | ₂Mutation | ₂Enzyme Inhibitors | ₂Fibroblasts | ₂Multiple Endocrine Neoplasia Type 1 | ₂Cell Separation | ₁Mutagenesis, Site-Directed | ₁Mutagenesis | ₁Retroviridae | ₁Gene Expression Regulation, Neoplastic | ₁bcl-2 Homologous Antagonist-Killer Protein | ₁Adenoviridae | ₁Cell Proliferation | ₁Tumor Necrosis Factor-alpha | ₁Cell Cycle | ₁DNA-Binding Proteins | ₁RNA, Messenger | ₁Nucleosomes

Xianxin Hua Unique Vocabulary (96) [single words, word pairs and phrases obtained by analysis of abstracts and titles, the leading number and the color conveys importance; Xianxin Hua might be a good expert witness on these terms]

₄₅menin | ₁₅men1 | ₁₄tumor | ₁₄cell | ₁₂proliferation | ₁₀suppressor | ₁₀dna | ₉apoptosis | ₈expression | ₈binding | ₈cells | ₇igfbp-2 | ₇gene | ₇endocrine | ₇multiple | ₆kinase | ₆menin-null | ₅nuclear | ₅complementation | ₅ask | ₄show | ₄essential | ₄protein | ₄type | ₄neoplasia | ₃activator | ₃function | ₃unclear | ₃s-phase | ₃direct | ₃promoter | ₃although
₁₂cell proliferation | ₁₀tumor suppressor | ₇suppressor menin | ₇multiple endocrine | ₆menin-null cells | ₅complementation menin-null | ₄binding dna | ₄endocrine neoplasia | ₄neoplasia type | ₄type men1 | ₄igfbp-2 gene | ₃s-phase kinase | ₃promoter igfbp-2 | ₃unclear menin | ₃activator s-phase | ₃syndrome characterized | ₃essential repression | ₃regulates expression | ₃menin essential | ₃men1 patients | ₃induces apoptosis | ₃tumor syndrome | ₃embryonic fibroblasts | ₃whereas complementation | ₃cells menin | ₃cooh terminus | ₃mutated men1 | ₃menin interacts | ₃murine embryonic | ₃wild-type menin | ₃menin regulates | ₃apoptosis murine
₇tumor suppressor menin | ₅complementation menin-null cells | ₄neoplasia type men1 | ₄endocrine neoplasia type | ₄multiple endocrine neoplasia | ₃activator s-phase kinase | ₃apoptosis murine embryonic | ₃menin essential repression | ₃tumor syndrome characterized | ₃menin-null cells menin | ₃murine embryonic fibroblasts | ₃mutated men1 patients | ₃whereas complementation menin-null | ₃induces apoptosis murine | ₃menin regulates expression | ₂cooh terminus menin | ₂unclear menin essential | ₂hereditary tumor syndrome | ₂suppressor menin activator | ₂direct binding dna | ₂expression insulin-like growth | ₂functional interaction tumor | ₂gene mutated men1 | ₂insulin-like growth factor | ₂factor binding protein | ₂menin activator s-phase | ₂encodes nuclear protein | ₂patients men1 encodes | ₂men1 patients men1 | ₂regulation cell proliferation | ₂binding dna tumor | ₂type men1 inherited

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