₄Joris A Veltman

Summary for 2004 [at-glance overview of Joris A Veltman work for the selected year as suggested by data mining algorithms]

This is an Authoratory overview of author Joris A Veltman. According to available data, in 2004 Joris A Veltman published at least 4 articles. This work was completed in collaboration with several authors including van Kessel, Ad Geurts and Schoenmakers, Eric F P M. Many other authors have collaborated with Joris A Veltman as well. There is no information about the funding or the grants awarded to support this work.

Joris A Veltman has rank 119 when compared to all other authors using the keyword Nucleic Acid Hybridization. Analysis of the article abstracts and the titles suggests that Joris A Veltman professional interests are focused around "comparative genomic hybridization", "array-based comparative genomic" and "genome-wide array-based comparative". These might also be referred to as "cell lines", "genomic hybridization" or "charge syndrome". Statistical analysis shows that Joris A Veltman's writing is likely to contains terms "loss", "cases", "chromosome", "pten", "lines" and "cell".

Unfortunately, there is not enough information to establish Joris A Veltman affiliation. Luckily, one email address is on file.

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Alternative names (1) [other authors with names similar to Joris A Veltman]

₁₆Veltman, J A

Year 2004 Charts [Joris A Veltman frequent coauthors and the most common unique vocabulary terms for the selected year]

Historical Performance Charts [graphical overview of Joris A Veltman publication and funding history for the range of years]

Emails (1) [email addresses extracted from Joris A Veltman affiliations]

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Joris A Veltman Coauthors (17) [all coauthors with the number of joint publications]

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₆van Kessel, Ad Geurts

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₅Schoenmakers, Eric F P M

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₁Vissers, Lisenka E L M

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Affiliations (3) [condensed summary of the Joris A Veltman affiliations, the leading number and the color conveys importance]

₁Department of Human Genetics, University Medical Center Nijmegen, PO Box 9101, 6500 HB Nijmegen, The Netherlands.
₁Department of Clinical Genetics, Lund University Hospital, Lund, Sweden.
₁Department of Pathology, Josephine Nefkens Institute, Erasmus Medical Center, Rotterdam, The Netherlands.

Key Opinion Leader In [Joris A Veltman is considered a key opinion leader in any keyword where the rank of 5 is achieved, the leading number before the keyword shows the author's rank as compared to all other authors]

₁₁₉Nucleic Acid Hybridization | ₇₂₂Oligonucleotide Array Sequence Analysis

Keywords (32) [keywords assigned to Joris A Veltman articles by PubMed, the leading number and the color conveys importance]

₄Humans | ₃Nucleic Acid Hybridization | ₃Oligonucleotide Array Sequence Analysis | ₂Cell Line, Tumor | ₂Chromosome Deletion | ₂Mutation | ₂Gene Deletion | ₁Gene Expression Regulation, Neoplastic | ₁Heart Defects, Congenital | ₁Genetic Markers | ₁Genes, Tumor Suppressor | ₁Coloboma | ₁Animals | ₁3' Flanking Region | ₁5' Flanking Region | ₁DNA-Binding Proteins | ₁DNA Helicases | ₁Magnetic Resonance Imaging | ₁Gene Amplification | ₁Infant | ₁Genes, Neoplasm | ₁Cerebral Cortex | ₁Choanal Atresia | ₁Child | ₁Transplantation, Heterologous | ₁Prostatic Neoplasms | ₁Mice | ₁Female | ₁Syndrome | ₁Male | ₁Chromosomes, Human, Pair 10 | ₁Homozygote

Joris A Veltman Unique Vocabulary (96) [single words, word pairs and phrases obtained by analysis of abstracts and titles, the leading number and the color conveys importance; Joris A Veltman might be a good expert witness on these terms]

₁₀loss | ₉cases | ₇chromosome | ₇pten | ₇lines | ₇cell | ₆pancreatic | ₆genomic | ₅deletions | ₅syndrome | ₅charge | ₅comparative | ₅hybridization | ₅homozygous | ₅gene | ₄around | ₄array-based | ₄small | ₄deletion | ₄targets | ₄amplification | ₄region | ₄genes | ₄flj11218 | ₄amplifications | ₄papss2 | ₄expression | ₄detected | ₃carcinoma | ₃genome-wide | ₃several | ₃major
₇cell lines | ₅genomic hybridization | ₅charge syndrome | ₅comparative genomic | ₅homozygous deletions | ₄array-based comparative | ₄around pten | ₃pancreatic carcinoma | ₃genome-wide array-based | ₃carcinoma cell | ₃alteration prostate | ₃cancer xenografts | ₃prostate cancer | ₃small region | ₃region around | ₃chromosome alteration | ₃loss small | ₃xenografts cell | ₃major chromosome | ₂amplification deletion | ₂chromodomain gene | ₂hybridization reveals | ₂detected flj11218 | ₂pancreatic carcinomas | ₂multiple amplification | ₂mutations member | ₂individuals charge | ₂member chromodomain | ₂family cause | ₂deletions pancreatic | ₂cause charge | ₂deletion targets
₅comparative genomic hybridization | ₄array-based comparative genomic | ₃genome-wide array-based comparative | ₃pancreatic carcinoma cell | ₃carcinoma cell lines | ₃major chromosome alteration | ₃alteration prostate cancer | ₃cancer xenografts cell | ₃loss small region | ₃region around pten | ₃small region around | ₃xenografts cell lines | ₃chromosome alteration prostate | ₃prostate cancer xenografts | ₂amplification targets novel | ₂member chromodomain gene | ₂hybridization reveals multiple | ₂homozygous deletions pancreatic | ₂gene family cause | ₂reveals multiple amplification | ₂amplification deletion targets | ₂copy number changes | ₂individuals charge syndrome | ₂novel homozygous deletions | ₂around pten locus | ₂mutations member chromodomain | ₂cause charge syndrome | ₂pten locus major | ₂chromodomain gene family | ₂locus major chromosome | ₂multiple amplification targets | ₂targets novel homozygous

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