₇E F Stange

Summary for 2004 [at-glance overview of E F Stange work for the selected year as suggested by data mining algorithms]

This is an Authoratory overview of author E F Stange. According to available data, in 2004 E F Stange published at least 7 articles. This work was completed in collaboration with several authors including Herrlinger, K and Fellermann, K. Many other authors have collaborated with E F Stange as well. There is no information about the funding or the grants awarded to support this work.

E F Stange has rank 40 when compared to all other authors using the keyword Crohn Disease. Analysis of the article abstracts and the titles suggests that E F Stange professional interests are focused around "patients nod2 mutations", "mutations crohn disease" and "inflammatory bowel diseases". These might also be referred to as "crohn disease", "nod2 mutations" or "hd-5 hd-6". Statistical analysis shows that E F Stange's writing is likely to contains terms "disease", "nod2", "crohn", "expression", "mutations" and "mucosal".

Unfortunately, there is not enough information to establish E F Stange affiliation. Luckily, several email addresses are on file.

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Alternative names (1) [other authors with names similar to E F Stange]

₅₃Stange, Eduard F

Year 2004 Charts [E F Stange frequent coauthors and the most common unique vocabulary terms for the selected year]

Historical Performance Charts [graphical overview of E F Stange publication and funding history for the range of years]

Emails (2) [email addresses extracted from E F Stange affiliations]

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Affiliations (6) [condensed summary of the E F Stange affiliations, the leading number and the color conveys importance]

₁Department of Internal Medicine I, Robert Bosch Hospital, Stuttgart, Germany.
₁Abteilung Innere Medizin 1, Zentrum für Innere Medizin, Robert-Bosch-Krankenhaus, Stuttgart.
₁Medizinische Klinik I mit Schwerpunkt Gastroenterologie/Infektiologie/Rheumatologie, Charité, Universitätsmedizin Berlin.
₁Robert-Bosch-Krankenhaus, Zentrum für Innere Medizin, Stuttgart.
₁Department of Gastroenterology, Hepatology and Endocrinology, Robert-Bosch-Hospital, Stuttgart, Germany.
₁Abteilung Innere Medizin I, Robert-Bosch-Krankenhaus, Stuttgart.

Key Opinion Leader In [E F Stange is considered a key opinion leader in any keyword where the rank of 5 is achieved, the leading number before the keyword shows the author's rank as compared to all other authors]

₄₀Crohn Disease | ₆₀Colitis, Ulcerative | ₇₆Remission Induction

Keywords (32) [keywords assigned to E F Stange articles by PubMed, the leading number and the color conveys importance]

₇Humans | ₄Crohn Disease | ₄Adult | ₃Colitis, Ulcerative | ₃Female | ₃Remission Induction | ₂Adolescent | ₂Pouchitis | ₂Diagnosis, Differential | ₂Inflammatory Bowel Diseases | ₂Anti-Bacterial Agents | ₂Recurrence | ₂Controlled Clinical Trials as Topic | ₂Gene Expression Regulation | ₂Risk Factors | ₂Immunity, Mucosal | ₂Middle Aged | ₂Male | ₁Colon | ₁Interleukin-8 | ₁Intestines | ₁alpha-Defensins | ₁Defensins | ₁Primary Prevention | ₁Thionucleotides | ₁Tumor Necrosis Factor-alpha | ₁Immunosuppressive Agents | ₁Tacrolimus | ₁Nod2 Signaling Adaptor Protein | ₁Azathioprine | ₁RNA, Messenger | ₁Magnetic Resonance Imaging

E F Stange Unique Vocabulary (96) [single words, word pairs and phrases obtained by analysis of abstracts and titles, the leading number and the color conveys importance; E F Stange might be a good expert witness on these terms]

₁₅disease | ₁₄nod2 | ₁₂crohn | ₈expression | ₈mutations | ₈mucosal | ₇defensins | ₅patients | ₅response | ₅hd-5 | ₅paneth | ₅bowel | ₅6-thioguanine-nucleotide | ₅levels | ₅diminished | ₄inflammatory | ₄bacterial | ₄diseases | ₄hd-6 | ₄barrier | ₄ileum | ₄human | ₄defensin | ₄pathogenesis | ₃cells | ₃colitis | ₃tioguanine | ₃lysozyme | ₃intestinal | ₃wild-type | ₃spla2 | ₃activity
₁₁crohn disease | ₅nod2 mutations | ₄hd-5 hd-6 | ₃patients nod2 | ₃inflammatory bowel | ₃paneth cells | ₃mutations crohn | ₃bowel disease | ₂mucosal barrier | ₂bowel diseases | ₂6-thioguanine-nucleotide levels | ₂may explain | ₂methylated 6-thioguanine-nucleotide | ₂hd-6 spla2 | ₂defensin expression | ₂ulcerative colitis | ₂wild-type nod2 | ₂mutations wild-type | ₂expression may | ₂card15 mutations | ₂nod2 card15 | ₂role defensins | ₂associated diminished | ₂disease associated | ₂thioguanine-nucleotides predict | ₂alpha-defensin expression | ₂alpha defensins | ₂pathogenesis chronic-inflammatory | ₂predict efficacy | ₂diminished mucosal | ₂mutations nod2 | ₂mucosal alpha-defensin
₃patients nod2 mutations | ₃mutations crohn disease | ₂inflammatory bowel diseases | ₂hd-5 hd-6 spla2 | ₂nod2 mutations wild-type | ₂expression may explain | ₂nod2 card15 mutations | ₂mucosal alpha-defensin expression | ₂efficacy tioguanine crohn | ₂defensins pathogenesis chronic-inflammatory | ₂chronic-inflammatory bowel disease | ₂predict efficacy tioguanine | ₂disease associated diminished | ₂crohn disease associated | ₂tioguanine crohn disease | ₂thioguanine-nucleotides predict efficacy | ₂associated diminished mucosal | ₂card15 mutations crohn | ₂role defensins pathogenesis | ₂pathogenesis chronic-inflammatory bowel | ₂diminished mucosal alpha-defensin | ₁thioguanine-nucleotide levels paradigm | ₁associated nod2 mutation | ₁nucleotide levels patients | ₁experienced bone marrow | ₁based consensus conference | ₁tioguanine therapy related | ₁patients immunohistochemistry identified | ₁results evidence based | ₁protozoa giardia lamblia | ₁mucosal adherent invasive | ₁crohn disease especially

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