₂Petronela Ancuta

Summary for 2004 [at-glance overview of Petronela Ancuta work for the selected year as suggested by data mining algorithms]

This is an Authoratory overview of author Petronela Ancuta. According to available data, in 2004 Petronela Ancuta published at least 2 articles. This work was completed in collaboration with several authors including Gabuzda, Dana and Mehle, Andrew. There is no information about the funding or the grants awarded to support this work.

Analysis of the article abstracts and the titles suggests that Petronela Ancuta professional interests are focused around "antiviral activity apobec3g", "tem cd16 monocytes" and "constitutive inflammatory conditions". These might also be referred to as "cd16 monocytes", "activity apobec3g" or "antiviral activity". Statistical analysis shows that Petronela Ancuta's writing is likely to contains terms "apobec3g", "cd16", "vif", "monocytes", "huvec" and "degradation".

The majority of Petronela Ancuta articles are affiliated with Department of Cancer Immunology and AIDS, Dana-Farber Cancer Institute, Harvard Medical School, 44 Binney Street, JFB 816, Boston, Massachusetts MA 02115, USA. Sadly, no email addresses were found for any of the affiliations.

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Alternative names (0) [other authors with names similar to Petronela Ancuta]

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Year 2004 Charts [Petronela Ancuta frequent coauthors and the most common unique vocabulary terms for the selected year]

Historical Performance Charts [graphical overview of Petronela Ancuta publication and funding history for the range of years]

Petronela Ancuta Coauthors (3) [all coauthors with the number of joint publications]

2

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Affiliations (1) [condensed summary of the Petronela Ancuta affiliations, the leading number and the color conveys importance]

₂Department of Cancer Immunology and AIDS, Dana-Farber Cancer Institute, Harvard Medical School, 44 Binney Street, JFB 816, Boston, Massachusetts MA 02115, USA.

Key Opinion Leader In [Petronela Ancuta is considered a key opinion leader in any keyword where the rank of 5 is achieved, the leading number before the keyword shows the author's rank as compared to all other authors]

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Keywords (32) [keywords assigned to Petronela Ancuta articles by PubMed, the leading number and the color conveys importance]

₂Humans | ₁Drug Interactions | ₁Gene Expression | ₁Gene Products, vif | ₁Brain | ₁Cell Line, Tumor | ₁Monocytes | ₁Tumor Necrosis Factor-alpha | ₁Half-Life | ₁Nucleoside Deaminases | ₁Receptors, Chemokine | ₁Lactones | ₁Immunosorbent Techniques | ₁Reverse Transcriptase Polymerase Chain Reaction | ₁Cells, Cultured | ₁Interferon-gamma | ₁Cell Adhesion Molecules | ₁Phosphorylation | ₁Recombinant Proteins | ₁Conserved Sequence | ₁Proteasome Endopeptidase Complex | ₁HIV-1 | ₁Inflammation | ₁Membrane Proteins | ₁Leupeptins | ₁Cysteine Endopeptidases | ₁Receptors, IgG | ₁Proteins | ₁Umbilical Veins | ₁Chemokines, CX3C | ₁Cysteine | ₁Transfection

Petronela Ancuta Unique Vocabulary (96) [single words, word pairs and phrases obtained by analysis of abstracts and titles, the leading number and the color conveys importance; Petronela Ancuta might be a good expert witness on these terms]

₁₅apobec3g | ₁₂cd16 | ₁₁vif | ₁₁monocytes | ₈huvec | ₈degradation | ₆fkn | ₅tem | ₅response | ₄monocyte | ₄activity | ₄tnf | ₄antiviral | ₄inflammatory | ₄ubiquitin-proteasome | ₄pathway | ₄conditions | ₄bmvec | ₃vascular | ₃constitutive | ₃results | ₃migration | ₃across | ₃soluble | ₃fractalkine | ₃transendothelial | ₃hiv-1 | ₂human | ₂overcomes | ₂innate | ₂onto | ₂produce
₁₀cd16 monocytes | ₄activity apobec3g | ₄antiviral activity | ₄ubiquitin-proteasome pathway | ₃constitutive inflammatory | ₃transendothelial migration | ₃tem cd16 | ₃inflammatory conditions | ₃monocytes response | ₃apobec3g degradation | ₂degradation apobec3g | ₂overcomes innate | ₂degradation ubiquitin-proteasome | ₂may contribute | ₂contribute pathogenesis | ₂ifn-gamma-stimulated huvec | ₂apobec3g promoting | ₂soluble fkn | ₂tnf ifn-gamma-stimulated | ₂vif overcomes | ₂promoting degradation | ₂innate antiviral | ₂fkn huvec | ₂migration cd16 | ₂fractalkine constitutive | ₂membrane-bound fkn | ₂monocytes across | ₂response fractalkine | ₂huvec bmvec | ₂response soluble | ₂expression membrane-bound | ₁microbial cellular
₄antiviral activity apobec3g | ₃tem cd16 monocytes | ₃constitutive inflammatory conditions | ₃cd16 monocytes response | ₂apobec3g promoting degradation | ₂overcomes innate antiviral | ₂innate antiviral activity | ₂may contribute pathogenesis | ₂degradation ubiquitin-proteasome pathway | ₂tnf ifn-gamma-stimulated huvec | ₂activity apobec3g promoting | ₂vif overcomes innate | ₂promoting degradation ubiquitin-proteasome | ₂fractalkine constitutive inflammatory | ₂expression membrane-bound fkn | ₂cd16 monocytes across | ₂response fractalkine constitutive | ₂fkn huvec bmvec | ₂transendothelial migration cd16 | ₂monocytes response fractalkine | ₂membrane-bound fkn huvec | ₂migration cd16 monocytes | ₁huvec brain microvascular | ₁inflammation involves monocyte | ₁mcp-1 triggered efficient | ₁proteins cd16 cd16 | ₁bmvec remained negative | ₁non-infectious hiv-1 mutations | ₁cd16 monocytes underwent | ₁onto vascular beds | ₁involves monocyte arrest | ₁degradation proteins cd16

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